Top 3 Vitamins You MUST take to Protect Your Kidneys!
If you’re a kidney disease patient, the right vitamins can make a huge difference in reducing creatinine and proteinuria naturally and slowing disease progression. In this video, we reveal the top 3 vitamins for kidney disease patients that help lower high creatinine levels, lower kidney inflammation, which can even help reverse CKD 2, 3, and 4. These essential vitamins, Vitamin B12, Vitamin D, and Vitamin B6, aid in kidney detox, lower blood pressure, and reduce the risk of CKD. Whether you have CKD, diabetic nephropathy, hypertensive nephropathy, autoimmune nephropathy, or nutrient deficiency-related kidney damage or are on dialysis, trying to prevent kidney failure, or looking for the best vitamins or supplements for kidney health, this video will help you. Watch now to learn how to protect your kidneys from failure naturally with the best foods for chronic kidney disease management!
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Time Stamps:
00:00 Intro
00:44 1. Vitamin D
07:56 2. Vitamin B6
16:05 3. Vitamin B12
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#kidneyvitamins #kidneyhealth #ckdvitamins
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Disclaimer:
The information provided in this video is for informational and educational purposes only and is not intended as a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of your physician or a qualified healthcare provider with any questions you may have regarding a medical condition. Never disregard professional medical advice or delay seeking it because of something you have seen or heard in this video. Reliance on any information provided in this video is solely at your own risk. If you are experiencing a medical emergency, please call your local emergency services immediately.
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Kidney Patients, Take These 3 Vitamins to Protect Your Kidneys!
Did you know that one in seven adults in the US has chronic kidney disease, CKD? If you’re one of them, managing your health goes beyond medications. It’s about nutrition, too. In this video, we’re uncovering the top three minerals every kidney patient should pay attention to. These minerals not only help support your kidney function, but can also play a critical role in preventing further damage. With the right balance, you can improve your quality of life, and even slow down kidney progression. We’ll dive into the science behind why calcium, magnesium, and phosphorus are crucial, what happens when you’re deficient, and the best foods and supplements to keep your levels in check. Don’t miss this. Your kidneys will thank you later. One, vitamin D. Vitamin D deficiency is extremely common in people with kidney disease, especially those with protein ura, elevated creatinine or diagnosed CKD or DKD. There are several natural mechanisms behind this. First, the kidneys convert inactive vitamin D, colocaliferol or erggoiferol into its active form, calcitriol 125 dehydroxy vitamin D. In kidney disease, especially in advanced stages, this conversion is impaired because of damaged renal one alpha hydroxilase activity. Second, protein ura leads to the loss of vitamin D binding protein DBP in urine which carries vitamin D in the blood. So even if you have vitamin D, it doesn’t reach target tissues well. Third, chronic inflammation in CKD disrupts vitamin D metabolism and increases fibroblast growth factor 23, FGF 23, which further suppresses the kidney’s activation of vitamin D. Also, limited sun exposure, darker skin, obesity, aging, and poor nutritional intake compound the deficiency. You can identify vitamin D deficiency by measuring serum 25 levels. In kidney patients, a level below 30 nanogs per milliliter is considered insufficient and below 20 nanogs per milliliter is deficient. Severe deficiency is below 10 nanogs per milliliter. Among CKD and diabetic kidney patients, over 70% are estimated to have suboptimal levels with up to 90% in late stage CKD. Symptoms may not show early but can include fatigue, bone pain, muscle weakness, low mood, poor sleep, and recurrent infections. Vitamin D deficiency affects multiple systems in the body. In kidneys, it worsens proteinura by increasing rayos activation and inflammation. It also accelerates CKD progression by enhancing fibrosis. For the heart and blood vessels, low vitamin D raises blood pressure and promotes left ventricular hypertrophy, endothelial dysfunction, and arterial stiffness, increasing the risk of heart failure and stroke. Bones suffer the most. Vitamin D is crucial for calcium absorption. and deficiency leads to secondary hyperarathyroidism causing bone demineralization, fractures and osteodistrophe. For nerves, low levels may contribute to neuropathy and poor nerve conduction. In the lungs, vitamin D plays a role in immunity. Deficiency increases respiratory infections and inflammation. Low levels are linked to increased anxiety and depression, especially in chronic illness. For weight, deficiency may worsen insulin resistance and is associated with higher BMI. For eyes, deficiency could increase risk of retinopathy in diabetics. Regarding fertility and libido, low vitamin D has been linked with lower testosterone in men and poorer egg quality in women. At the cellular level, vitamin D acts via the vitamin D receptor, VDR, which is expressed in kidney cells, cardiac meioytes, immune cells, and more. When activated, it regulates hundreds of genes. In kidney disease, VDR activation reduces inflammation by suppressing NFCappa B fibrosis via TGF beta and slows the progression of glomemeular damage. It also helps reduce RAS overactivity, a major player in both kidney and cardiovascular issues. Top five natural sources of vitamin D with portion sizes suited for CKD patients include cod liver oil. 1 tsp equals approximately 1,360 IU or 227% of RDA. Salmon 85 gram cooked equals approximately 570 IU or 95%. Mackerel 85 gram equals approximately 570 IU or 95%. Approximately 360 international units or 60%. Egg yolk one large equals approximately 40 international units or 7%. and sardines. Two fish equals approximately 46 IU or 8%. These are high quality sources but need moderation due to phosphorus or sodium in some of them. Among luxury recipes with high vitamin D content, salmon on 150 g salmon fillet baked in puff pastry offers approximately 600 IU. Miso glazed black cod 120 grams cod provides approximately 500 IU. Sea urchin uni rato 100 g of fresh sea urchin can have approximately 400 IU although highly seasonal and expensive for CKD. Portion control and phosphate management are essential. Bioavailability from foods depends on fat content. Vitamin D is fat soluble, so it absorbs best with dietary fat. Cooking methods matter too. Grilling or baking preserves more vitamin D than boiling. Also, cod liver oil has near complete bioavailability when taken with food. For supplements, two of the best formulas are one collicaliferol D3 soft gels 5,000 IU daily dose around $15 to $20 for 120 soft gels. Two calcitrial capsules, the active form used in CKD stages 4 to 5, commonly prescribed in microgram doses like 0.25 mg daily. priced at 25 to $40 per 30 capsules. You should choose D3, not D2, unless advised otherwise as it’s more effective and longerlasting. A good formula will have third party testing like USP or NSF certification, minimal additives, and proper labeling. Avoid supplements with megaoses unless directed. High doses without monitoring can cause hypercalcemia, especially in CKD. While supplementing, avoid over-the-counter calcium supplements unless prescribed. This can lead to calcium overload. Foods that increase deficiency risk include high fitate grains, unsoaked bran, unprocessed whole grains, which reduce absorption, and processed junk food that worsens obesity and inflammation. Avoid pairing with bile acid sequesterance or highdosese cortosteroids which reduce absorption. High oxilate greens like spinach block calcium and vitamin D synergy. So they’re not ideal when eaten in excess. Two, vitamin B6. Vitamin B6 or pyodoxine plays a central role in many bodily systems and becomes especially important in patients with kidney disease. Whether that’s proteinura, elevated creatinine, chronic kidney disease, CKD or diabetic kidney disease, DKD, naturally occurring deficiency in these patients happens due to a few interconnected mechanisms. First, kidney disease causes urinary loss of vitamin B6 and its active co-enzyme form. Pyodoxal 5 prime phosphate or PLP which is water soluble and easily lost during protein ura and dialysis. Second, inflammation and oxidative stress in CKD deplete vitamin B6 faster and the impaired renal activation of B6 reduces its conversion to the bioactive paradoxal phosphate form. Third, poor dietary intake, nausea, dietary restrictions, and low protein renal diets reduce B6 intake from food. Drugs used in kidney patients like hydrolysine, isazid or loop diuretics can further interfere with absorption or increase its breakdown. Deficiency is identified through blood measurement of pyodoxal phosphate levels. Normal PLP ranges are 5 to 50 micrograms per liter and anything under 20 micrograms per liter is often considered suboptimal with less than 10 micrograms per liter indicating a clear deficiency. Studies show that 35 to 60% of dialysis patients and over 25% of p-ialysis CKD patients have low B6 levels. Clinical signs can be subtle or severe like fatigue, nerve tingling, depression, anxiety, low appetite, irritability, and glossitis. In advanced stages, deficiency worsens neuropathy and anemia already common in kidney disease. This deficiency affects major systems. In the kidneys, it weakens antioxidant defense and worsens inflammation and oxidative damage, accelerating CKD progression. In the heart and vessels, B6 deficiency raises homocyine levels, promoting atherosclerosis, stiff arteries, hypertension, and increased cardiovascular mortality risk. Already a leading cause of death in CKD. For bones, B6 is needed for collagen cross-linking and lysol oxidase activity. Its deficiency contributes to weak bones and worsens renal osteodistrophe. In nerves, low B6 impairs GABA synthesis causing neuropathy, burning feet, confusion, and sleep disturbances. In lungs, B6 is needed for immune defense and reducing inflammatory cytoines. deficiency increases the risk of infection. Low levels of B6 worsen mood by reducing dopamine and serotonin production, increasing the likelihood of depression and anxiety. It can affect weight indirectly through appetite loss and metabolic disruption. B6 supports eye health via its role in homocyine metabolism. Deficiency may worsen retinal microvascular changes in diabetes. As for fertility and libido, B6 is required for hormonal balance. Deficiency can lower testosterone and disrupt estrogen metabolism, potentially reducing libido and causing cycle irregularities in women. At the cellular level, vitamin B6 mainly as PLP acts as a co-enzyme in over 100 enzyatic reactions. These include amino acid metabolism, neurotransmitter synthesis like serotonin, GABA, dopamine, heynthesis for red blood cells, and the regulation of inflammatory pathways. In kidney disease, B6’s antioxidant role helps reduce reactive oxygen species and suppress NFKB mediated inflammation. It also regulates homocyine by acting with folate and B12. Without enough B6, homocyine levels rise, which accelerates glomemeular and vascular damage. Top five natural B6 sources suitable for CKD include bananas. One medium banana equals 0.4 mg equals 30% RDA boiled potatoes. One medium= 0.7 mg= 50% RDA. Fortified oats 1 cup equals 0.5 mg equals 35% RDA. Cooked spinach 1 cup= 0.4 mg equals 30% RDA. And chicken breast 85 g= 0.5 mg= 35% RDA. These are safer for moderate CKD patients because they are relatively low in potassium and phosphorus when portioned correctly. In diialysis patients, portion control is key to manage potassium load. Three luxury global dishes rich in B6 include Japanese grilled yellow fin tuna steak. 170 g equals approximately 1 mg B6 equals 70% RDA. Moroccan spiced roasted chicken thighs with lentils, 200 g equals approximately 1.2 mg equals 85% RDA. and Peruvian quinoa stuffed bell peppers with mushrooms. 250 grams equals approximately 0.9 milligrams equals less than 1% RDA. 65% RDA. All these combine high protein B6 sources and are often used in nutrition therapy for keptic CKD patients to support appetite and energy balance. B6 bioavailability from animal sources like chicken, fish, and eggs is higher, 75 to 100%. Compared to plant sources, 45 to 75%. Because PLP is proteinbound and better absorbed from animal proteins. However, heat and long cooking times can degrade B6, so light steaming or roasting is preferable. Two highquality B6 supplement formulas include Solar Vitamin B6 100 mgram tablets. Price approximately $10 for 100 tablets. And now foods P5P 50 mg. Pyodoxal 5 phosphate form better absorption. Price approximately $12 for 90 tablets. The latter is especially useful in CKD due to impaired B6 activation. A good supplement will state the form P-5-P is better than pyrooxine HCl for CKD. Have third party testing or USP certification and avoid unnecessary additives or magnesium sterate. Poor quality ones often use high pyodoxin doses without co-enzymes which can cause toxicity and imbalance. Precautions. Long-term use of high doses greater than 200 mg per day can cause sensory neuropathy. Always monitor blood levels during supplementation. Some drugs like isoniaid, hydrolysine, and penicyamine increase B6 need. Foods that increase deficiency include high oxilate vegetables, spinach, beet greens that bind PLP, and excessive alcohol, which depletes B6 stores. Bad food pairings include very high protein meals taken with raw egg whites. Avidin inhibits B6 indirectly or ultrarocessed snacks with high sugar and preservatives that deplete B6 over time. Three, vitamin B12. Vitamin B12 deficiency in kidney disease patients, especially those with conditions like protein ura, elevated creatinine, chronic kidney disease, CKD, or diabetic kidney disease, DKD, can be quite common due to several interconnected factors. Kidney dysfunction can affect the body’s ability to process vitamin B12, partly due to impaired renal clearance and disruptions in protein metabolism. Additionally, certain medications commonly used to manage kidney disease like proton pump inhibitors, PPIs, or metformin can interfere with vitamin B12 absorption. Patients with CKD often have reduced intrinsic factor, a protein required for B12 absorption in the intestines, which further exacerbates the deficiency. The symptoms of B12 deficiency can manifest across various body systems, including the kidneys, heart, blood vessels, and more. In the kidneys, a deficiency can worsen renal function by impairing methylation processes and increasing homocyine levels, which in turn leads to further damage to kidney tissue. As for the heart and blood vessels, elevated homocyine levels due to vitamin B12 deficiency are a known risk factor for cardiovascular diseases as they promote the formation of plaques in the blood vessels leading to higher blood pressure and increasing the risk of strokes and heart attacks. In terms of bones, B12 plays a key role in the synthesis of red blood cells and maintaining bone health and its deficiency can lead to osteoporosis or fractures. Nerve function also suffers with symptoms like numbness, tingling and neuropathy. The central nervous system is particularly sensitive to low B12 levels leading to mood disorders such as anxiety or depression. Patients may also experience weight loss due to a decrease in appetite and overall fatigue. The eyes can be affected by vision problems as B12 deficiency can cause optic neuropathy. For fertility and libido, insufficient B12 can disrupt hormone levels, potentially leading to infertility or decreased libido. Identifying B12 deficiency is typically done through blood tests measuring serum vitamin B12 levels. A normal B12 range is typically between 200 to 900 pogs per milliliter and values lower than 200 pogs per milliliter often indicate deficiency. However, in kidney disease patients, these values can sometimes be misleading and additional tests such as methylalonic acid, MMA or homocyine levels might be more accurate indicators of a deficiency as these metabolites accumulate when B12 is low. Early identification is crucial as prolonged deficiency can lead to irreversible nerve damage and further kidney impairment. In terms of natural food sources, some of the top sources of vitamin B 12 for kidney disease patients include animal-based products, though caution should be exercised with portion sizes due to the potential strain they can place on the kidneys. For instance, a 3oz portion of cooked liver provides about 1,200% of the daily recommended intake of B12, while a 3oz portion of cooked fish like salmon can offer about 80% of the daily requirement. Eggs, too large, contain about 46% of the daily B12 requirement, and dairy products like milk or yogurt can provide around 18 to 20% per cup. Shellfish like clams are another potent source providing more than 100% of the daily need in just a 3 oz serving. In kidney disease, balancing protein intake is critical, so portion sizes should be smaller to prevent further kidney strain. When it comes to luxury foods with the highest B12 content, fuera, caviar, and lobster are notable examples. A 1 oz serving of fuigra offers over 200% of the daily B12 intake while caviar provides about 80% and lobster offers about 50%. These foods are not only nutrientdense but also expensive and less commonly consumed making them accessible in luxury recipes or high-end meals. However, the bioavailability of B12 in these foods is quite high, meaning the body can absorb and utilize it efficiently. Supplements are often necessary for kidney disease patients who cannot maintain sufficient B12 levels through food alone. Two of the best available supplements are methylcobalamin and cyanocobalamin. Methylcobalamin is a more bioavailable form and is recommended for kidney disease patients because it doesn’t require the liver to convert it as cyanocobalamin does. Typical doses for kidney disease patients are around 500 to 1,000 micrograms daily depending on the severity of the deficiency. Prices for these supplements can range from $15 to $30 for a month supply depending on the brand. A good supplement should contain no artificial colors or preservatives, and it’s essential to choose a reputable brand with third party testing for purity. Poor formulas may contain inactive ingredients or too much filler, which can be harmful, especially in kidney disease patients. Certain foods can exacerbate vitamin B12 deficiency, especially those that interfere with B12 absorption or increase the body’s demand for it. For example, excessive alcohol intake can impair B12 absorption and high-fiber diets or foods containing high amounts of phitates or calcium can reduce B12 bioavailability. Furthermore, taking certain medications like metformin for diabetes or proton pump inhibitors for acid reflux can impair B12 absorption and worsen deficiency in kidney disease patients. Avoiding certain food pairings can also be helpful, such as consuming high-fiber meals with B12 rich foods, which can hinder the vitamin’s absorption. It’s better to take B12 supplements separate from meals that contain large amounts of fiber or calcium to maximize absorption. If you found this video helpful, make sure to hit that like button and subscribe to stay updated on more tips for managing kidney health. Don’t forget to share this with anyone who could benefit from this information. If you have any questions or want to share your thoughts, drop a comment below. I’d love to hear from you.